Veteran's Consensus Statement on Post-Cycle Recovery

[w_llewellyn]

<BLOCKQUOTE class="**-ubbcode-quote"><font size="-1">quote:</font><HR>Originally posted by TonyDelk:
Thanks for your reply's Bill.

One question for ya. If we acknowledge what you're saying is true what about the *proposed logic* of using Clomid/Arimidex together at higher than clinical doses for 3-5 days every 4th week while on cycle to maintain testicular volume?

While surely this won't solve the problem of testicular atrophy, won't it help reduce it, thereby avoiding the complete crash that most AAS users experience post cycle when starting HPTA resotoration therapy?

Thanks for your response.

TD<HR></BLOCKQUOTE>

Probably not, because the androgens will be more than sufficient to shut off gonadotropins. The only thing that would really works, at least in theory, is the periodic use of HCG, which would help maintain testicular mass while endogenous LH is diminished. But there are a lot of issues with using it for longer periods, so I am not a big fan of it (in this context) myself.

[archive_macrophage69alpha]

<BLOCKQUOTE class="**-ubbcode-quote"><font size="-1">quote:</font><HR>Originally posted by w_llewellyn:
<BLOCKQUOTE class="**-ubbcode-quote"><font size="-1">quote:</font><HR>Originally posted by macrophage69alpha:
btw- as this is an ongoing travesty... clomifin and nolvadex are NOT ANTI-ESTROGENS.. they are SERMS.. selective Estrogen Receptor Modulators.. they can both "activate" and "block" the ER.. dependent on tissue..(as well as the serm- of which these are but two among many) and in some tissues they have high and some low affinity.. (some virtually none)

MP<HR></BLOCKQUOTE>

Enough whining. Everyone knows they are SERMs. Not every little detail needs to be explained in every article that mentions Nolvadex or Clomid. "Anti-estrogen" is a fully sufficient description unless you want to get into an indepth discussion of its partial agonistic and antagonistic activity. Geez you are a pedantic twit sometimes.<HR></BLOCKQUOTE>

actually.. no they dont... would venture to guess that 19 out of 20 people that read your articles have no idea what a SERM is. Or the importance of the fact that they are mixed agonist/antagonists of the ER.

the issue is that by using a term like "anti-estrogen", while you understand the difference, perpetuates the misperception that clomifin and nolvadex act solely as "estrogen-blockers" which is not the case.

there are other impacts of SERMS particularly with respect to adiposity and mental health where the mixed agonist/antagonist nature has great impact-- often deleterious.

MP

[w_llewellyn]

<BLOCKQUOTE class="**-ubbcode-quote"><font size="-1">quote:</font><HR>Originally posted by macrophage69alpha:
[quote]Originally posted by w_llewellyn:
[quote]Originally posted by macrophage69alpha:
actually.. no they dont... would venture to guess that 19 out of 20 people that read your articles have no idea what a SERM is. Or the importance of the fact that they are mixed agonist/antagonists of the ER.

the issue is that by using a term like "anti-estrogen", while you understand the difference, perpetuates the misperception that clomifin and nolvadex act solely as "estrogen-blockers" which is not the case.

there are other impacts of SERMS particularly with respect to adiposity and mental health where the mixed agonist/antagonist nature has great impact-- often deleterious.

MP<HR></BLOCKQUOTE>


Stop being such a drama [star] Macro.

[This message was edited by Ulter on 07-15-2003 at 09:56 AM.]

[archive_Ulter]

Let's put the name calling away please.

[archive_Mr. Nobody]

Recap:

Take out Androgel since it may be detrimental to the HPTA (inconclusive at the least)

Take out Bromocriptine since it may not be needed

Add in nootropics at end

Problem:
Testicular atrophy needs to be rectified or avoided. Use of clomid while on AAS therapy may not prevent atrophy due to the androgen feedback. Minimize excessive LH saturation during cycle.
Solution:
Leave clomid for post cycle
Use H C G in bursts
Avoid prolonged cycles

Below find first revision to this CS:

Veterans’ Consensus Statement on Post-Cycle Recovery REVISED

Anabolic/androgenic steroids are used widely in human and veterinary medicine, and are increasingly useful to the training methods of elite athletes. Benefits of the intelligent use of anabolic/androgenic steroids include enhanced quality of life and the promise of greater longevity, as well as marked improvements in body composition, strength, and stamina. However, anabolic/androgenic steroids produce their benefits by interfering with the endocrine system, a complex system of glands and brain structures that are normally kept in an homeostatic state of balance by the action of countless subtle, sensitive feedback mechanisms. The perturbation in normal endocrine function that is introduced by the use of anabolic/androgenic steroids can, through these feedback mechanisms, elicit compensatory endocrine responses, such as up- or down-regulation of essential enzyme stores or of receptor molecules, in order to maintain homeostasis. When these compensatory mechanisms persist into the post-cycle era after steroids have been withdrawn, unwanted effects can occur, such as fatigue, depression, loss of sex drive, loss of size and strength, and others. Fortunately, both prophylactic and restorative measures that the athlete can take in this situation are now fairly well known.

Many athletes have agreed that androgenic/anabolic steroids render appreciable gains for a limited time only. As said gain period differs between individuals, this CS will refrain from any recommendations to the optimum time of such therapy but discuss methods of restoring optimum normal endocrine function.

It should be noted that the longer a cycle lasts past the eight-week mark, the harder testosterone recovery becomes. The best way of gauging ones hormonal milieu and planning compensatory measures is to have blood tests done prior to and following cessation of AAS therapy. For the purpose of this Consensus Statement and the awareness of a lack of testing athletes, the following universally accepted post cycle hormone status is assumed:

a) Luteinizing Hormone (LH): low to none, Luteinizing Hormone Releasing Hormone (LHRH): low to none
b) Testosterone (T): low
c) Estrogen (E): high in relation to T
d) Cortisol (C): high
e) Red Blood Cell (RBC) count: falling


While all of these hormone measurements are assumed on the low end of the scale, biochemical individuality will ultimately determine where a person’s levels fall. So assumption of low to substandard levels will not always be true in everyone.


1. What are the goals of testosterone recovery?

The return of hormonal balance is but one goal of this program. To create a transitional period of minimized muscle loss and sustained and/or increased motivation is another.


2. Detailed Recommendations

If the athlete is ready to come off and is still taking long acting esters he shall switch to short acting drugs in order to have complete control of exogenous hormone levels. A “waiting period” for esters to clear is unacceptable and provides for a slow slide into the post cycle catabolic state. This period of short acting supplements shall last for a minimum of 2 weeks.

a) Luteinizing Hormone and shrunken testicles

H C G
If the testis have atrophied, the introduction of H C G at 1000iu x 14 days is necessary. To prevent this atrophy from happening, the use of H C G at 1000iu x 7 days every fourth week of the cycle is recommended. This will provide exogenous LH and must only be used to restore/keep proper testicle size.
Week 1-2: H C G, 1000iu ed

b) Low testosterone and lack of motivation

The introduction of exogenous hormones to compensate for the low endogenous testosterone levels may help to keep loss of drive, strength and muscle at bay but may also slow the recovery process. The below drug and application was chosen for its limited impact on the HPTA

D i a n a b o l
Studies and empirical evidence have shown Dianabol to be beneficial to keep Cortisol in check and provide some intermediate relief from the symptoms of low testosterone via an increase of dopamine, IGF-1, and Central Nervous System stimulation. The heightened dopamine will combat Prolactin and help raise the levels of endogenous Human Growth Hormone. Other studies point to a lack of LH suppression when taken first thing in the morning. It shall be noted that only a low dose upon rising is recommended in order to avoid further disruption of the HPTA
Week 1-6: 10mg dbol am, ed

c) High Estrogen and suppressed Hypothalamus- Pituitary- Testicular- Axis (HPTA)

Estrogen acts as the primary messenger of testosterone production. Testosterone is aromatized into estrogen, which signals the Hypothalamus to stop producing the proper testosterone release hormones. Estrogen must be kept low.

A r i m i d e x
A powerful aromatize inhibitor shall be part of every cycle. For testosterone recovery it is used to keep the testosterone/ estrogen balance in favor of testosterone. It is also of help to keep any additionally occurring estrogen from dbol and Androgel low to none. Studies have shown a 54% increase of testosterone in eugonadal patients
Week 1-10: ½-1mg ed
C l o m i d
Universally accepted as THE testosterone recovery tool. It blocks estrogen from the HPTA and stimulates the production of LHRH. LHRH then initiates the production of LH, which in turn signals the testis (if not atrophied) to produce testosterone.
Week 3-5: 100mg ed
Week 6-8: 50mg ed

d) High Cortisol, suppressed HPTA and catabolism

Cortisol is catabolic. It is the enemy of all anabolism and must be kept in check. While it is blocked when under the influence of AAS, it is free to attach to the Anabolic Receptors (AR) once the steroids leave. Due to this blockage Cortisol tends to accumulate and increase when on. A low level is desirable however since it is important for other vital functions such as control of inflammation. Balance is the key.

V i t a m i n C
At 3-5g before heavy workouts, it keeps the exercise induced rise of Cortisol in check
Always: 3-5g before workouts
D H E A
A useless pro-hormone as far as anabolism is concerned, this substance is great to keep Cortisol within normal levels. There is a correlation between high Cortisol and low DHEA levels.
Week 1-6: 150mg am and pm
D e x t r o s e a n d M a l to d e x t r i n
It is neither a supplement nor a drug, but these carbohydrates have a very high glycemic index and keep Cortisol levels low by increasing insulin. They also provide excellent energy for heavy workouts. In order to not gain unwanted fat, dextrose and/or Maltodextrin shall be ingested during your workout and with your post workout shake only.
Always: 100g with workout water and 100g with post workout shake

e) Red Blood Cell Count and Stamina

C r e a t i n e
The use of Creatine has shown to increase ATP metabolism and cellular water storage among many other things. This is very beneficial because it provides for heightened nutrient storage and a slight increase in anabolism as well as workout stamina. Perfect with dextrose/Maltodextrin.
Always: 5g with workout water and 10g with post workout shake
V i t a m i n B - 1 2 & I r o n
Prolongs the life of your RBC and may be beneficial for increased oxygen transport
Week1-8: 1,000mcg ed

Miscellaneous beneficial drugs, supplements and recommendations

Z i n c
Assists with testosterone production and is always low in weight lifting subjects. Do not consume with calcium for ease of absorption
Week1-8: 50mg ed
M a g n e s i u m
Has too many benefits for weight lifters to list
Week 1-8: 800mg every evening
V i t a m i n B - 6
Assists with testosterone production, keeps Prolactin in check and is very relaxing
Week 1-8: 200mg every evening
M e l a t o n i n
May improve sleep pattern and help increase HGH. With this supplement, the less you take the more it works.
Always: 1.5mg at nite
D e p r e n y l
Known as one of the most favorite life extension drug this dopamine enhancer provides anti-depressant properties as well as possible IGF-1 increase. Do not take with Bromocriptine.
Week 7 & 8: 5mg eod in the morning
N o o t r o p i c s
A course of these "smart drugs" may be beneficial to improve blood flow to the brain and HP. No specific drug, combination of drugs and/or drug course recommendations shall be made due to varying individual preferrences
W o r k o u t a n d c a l o r i c r e s t r i c t i o n
Workouts shall be brief and focus on retaining your newly gained strength. A power lift routine may be advantages at this stage. Calorie intake shall match expenditure; a calorie-restricted diet shall commence only upon complete recovery of natural testosterone production.


3. Final word

This program is based on empirical evidence, research and experimentation and represents the maximum effort to recover one’s testosterone production. Some of the above supplements and drugs may not be required or may not agree with every individual and advances in medicine may provide newer and more useful drugs for the testosterone recovery following steroid therapy.
Furthermore, it must be noted that a period of 8 weeks of abstinence from all drugs (vitamins and supplements excluded) is the minimum time recommended and that a blood test to assess actual testosterone recovery act as the only gauge for the timing of the next hormone therapy.

Anabolic/androgenic steroids wisely used have many benefits, but they produce their benefits by perturbing the natural course of endocrine function, something that can have consequences for the athlete in terms of enduring dysregulation of said endocrine function upon the cessation of anabolic use. Fortunately, both prophylactic and restorative measures that the athlete can take to restore endocrine function and prepare the way for the next cycle of anabolics are fairly well known. Problems and their solutions include (a) low levels of Luteinizing Hormone and shrunken testicles, treated by H C G, (b) low testosterone and lack of motivation, treated by Dianabol morning applications, (c) high estrogen and suppressed Hypothalamus-Pituitary-Testicular Axis (HPTA) function, treated by Arimidex and Clomid, (d)high Cortisol, suppressed HPTA and catabolism, treated by Vitamin C, DHEA, dextrose and Maltodextrin, and (e) suppressed red blood cell count and reduced stamina, treated by Creatine, Vitamin B-12 and iron. In addition, a variety of miscellaneous beneficial drugs and supplements, such as zinc, magnesium, Vitamin B-6, Melatonin, Deprenyl and misc. Nootropics can speed post-cycle recovery.

Disclaimer:
Mr. Nobody is presenting fictitious opinions and does in no way, shape or form encourage, use nor condone the use of any illegal substances or the use of legal substances in an illegal manner.
The information discussed is strictly for entertainment purposes only and shall not take the place of qualified medical advice.

[This message was edited by Mr. Nobody on 07-17-2003 at 02:26 PM.]

[archive_TonyDelk]

Expect further revisions. It's been tough trying to get a hold of Bill Roberts so that his input could be scrutinized.

Ulter where is Dr. Scruggs?

In my experience, H C G 's usefulness lessens the more it is used. So I'm not down with it 7x every fourth week. The Clomid/Arimidex protocol works well for me and I will admit that I gleaned that from Animal.

BR use to advocate Clomid all the way through cycles to prevent testicular atrophy and also to help with agression problems. He has since softened his stance but I'd like to still see him chime in here.

If anybody can get the word out to him, please do. The email addy's I use to use to communicate with him are no longer any good.

Fine thread brothers.

TD

[archive_Ulter]

I think that c) Estrogen (E): high
Should be E high in relation to T. Because 2 weeks off the cycle E is much lower but not in the T/E ratios. Because that looks like we're saying E is raised on it's own somehow post cycle and it's not.

[This message was edited by Ulter on 07-16-2003 at 11:07 AM.]

[Q]

I think you should add a few more areas.

The Catabolic state:
You have covered the Endocrine recovery part in detail but no matter how perfect our endocrine recovery is we will still have a few weeks of a non-anabolic / potentially catabolic state to deal with. During this time I feel it is a best practice to use non-androgenic anabolics like HGH and Insulin. Also a change in workout frequency and or intensity may be in order since overtraining can become a problem.

Mental State:
I feel you should also add a little more on how to deal with depression, lack of sex drive, Lack of motivation to lift and so on.

Besides that it looks like it is coming togather.
Good job!

Q

[archive_Mr. Nobody]

Good point Q,
I originally had insulin and EPO included as non-androgenic anabolics but felt their inherent danger to be somewhat risky for a Consensus Statement. HGH is very expensive still, IGF-1 R3 is still unproven.
However, if the majority agrees, I will find a way to include the above.
Depression and lack of sex drive/motivation and so on were covered with the morning dbol....and Androgel, which after further research I am leaning towards including once more. If there are other meds with low side effects to mitigate those conditions, let me know.

Excellent point on the workout frequency...maybe even rest for a week

Disclaimer:
Mr. Nobody is presenting fictitious opinions and does in no way, shape or form encourage, use nor condone the use of any illegal substances or the use of legal substances in an illegal manner.
The information discussed is strictly for entertainment purposes only and shall not take the place of qualified medical advice.

[archive_the truth]

I agree with Q as well, particularly with regard to training frequency , intensity and duration. I usually take a week or two off while recovering.

[Fonz]

Looking better.

But remember, this is old science.

What about GH? Insulin? Like Q said. They offer MANY post-cycle benefits when added to a normal post-cycle recovery program. You should put a high-tech version up as well.

When i come off(3 years this coming Feb), I'm going to be using:

1. 20mcg IGF-1 R3 Long (receptor Grade) 25 days on/ 25 days off and repeat. Total 100 days(About 14 weeks).(2mg total IGF-1 R3 Long)
This will spare LBM, while stopping BF% accrual due to low test levels. I might even out on some muscle. IGF-1 R3 Long has also been shown to up-regulate LH and testicular size.
2. Androgel for the first 4 weeks to keep my libido going. This gives H C G the time to get my endo test churning again.
3. H C G at 5000mg/week for the first 4 weeks
4. Clomid at 100mg/day for the first 4 weeks
5. Arimidex at 1mg/day for the first 4 weeks followed by 10 weeks of Aromasin at 25mg ED.

I have found letrozole to destroy my sex drive at 2.5mg ED.

The clomid will keep my lipid levels good while on arimidex. Then when I switch to aromasin, they will remain stable. Aromasin does not really affect Lipid levels.

Total recovery time = 14 weeks.

Fonz

"Great minds talk about ideas, average minds talk about facts, and weak minds talk about people"

---- Fonz 6/2002

[archive_Mr. Nobody]

The reason its old science is because I actually tried everything proposed and did not just relay something I read somewhere

I might put insulin back under Cortisol protection and EPO under red blood cell increase (even though I emphasized malto for endo slin and B-12 for RBC). However, I have no experience with HGH, but based on research, you need months for the benefits to appear. IGF-1 R3, a couple of my friends took and they felt it wasn't worth the money (50mcg ed)....again its good to have first hand experience. So with IGF-1 R3 its hit or miss......

Fonz, H C G and Clomid at the same time is counterproductive IMO, H C G supplies LH, while Clomid is supposed to elicit LH secretion

Also keep in mind, this CS was supposed to address mostly testosterone recovery

Another revision will be upcoming soon

Disclaimer:
Mr. Nobody is presenting fictitious opinions and does in no way, shape or form encourage, use nor condone the use of any illegal substances or the use of legal substances in an illegal manner.
The information discussed is strictly for entertainment purposes only and shall not take the place of qualified medical advice.

[Q]

Talking about Insulin is a must in my book. You can give two paths to its use. Insulin use and manipulation of insulin through diet and supplements. As for HGH, I agree that long term use is needed to see it work in an already highly anabolic environment but we are not trying to gain muscle here, just to keep hold of it. I agree its cost is limiting but if you have the means it is a good idea especially combined with insulin. And of course insulin should be combined with Creatine.

As for the this CS being mainly about Test recovery, Why limit it?

Q

[DaMan]

<BLOCKQUOTE class="**-ubbcode-quote"><font size="-1">quote:</font><HR>Originally posted by Q:

As for the this CS being mainly about Test recovery, Why limit it?

Q<HR></BLOCKQUOTE>

Yea it does say POST-CYCLE RECOVERY, not post-cycle TEST recovery

Fonz, on a nother note, do you really think it's necessary to run aromatase inhibitors for 14 weeks?

[SteveDFW]

What about ephedrine and/or clen for their anti-catabolic abilities?

[archive_Ulter]

I agree that Post Cycle recovery is what should be covered and that all aspects of that could be addressed. Including the use of insulin, clen, etc. with the proper cautionary wording.

[archive_Bjaarki]

I agree with Q, Ulter and the other bros that the statement should be as wide-ranging as necessary, including the use of the agents just mentioned, and with recommendations on training layoffs, etc.. This will be a tough job to integrate all this, Mr. N., but the result will without a doubt justify the effort. This thread has already attracted more pointed commentary than did either of the other two Consensus Statements. That's a great measure of its perceived importance.

Nice work!

Bjaarki

[archive_Mr. Nobody]

Recap:

Add insulin
Add EPO
Add Ephedrine/Clen


Below find second revision to this CS:

Veterans’ Consensus Statement on Post-Cycle Recovery

Anabolic/androgenic steroids are used widely in human and veterinary medicine, and are increasingly useful to the training methods of elite athletes. Benefits of the intelligent use of anabolic/androgenic steroids include enhanced quality of life and the promise of greater longevity, as well as marked improvements in body composition, strength, and stamina. However, anabolic/androgenic steroids produce their benefits by interfering with the endocrine system, a complex system of glands and brain structures that are normally kept in an homeostatic state of balance by the action of countless subtle, sensitive feedback mechanisms. The perturbation in normal endocrine function that is introduced by the use of anabolic/androgenic steroids can, through these feedback mechanisms, elicit compensatory endocrine responses, such as up- or down-regulation of essential enzyme stores or of receptor molecules, in order to maintain homeostasis. When these compensatory mechanisms persist into the post-cycle era after steroids have been withdrawn, unwanted effects can occur, such as fatigue, depression, loss of sex drive, loss of size and strength, and others. Fortunately, both prophylactic and restorative measures that the athlete can take in this situation are now fairly well known.

Many athletes have agreed that androgenic/anabolic steroids render appreciable gains for a limited time only. As said gain period differs between individuals, this CS will refrain from any recommendations to the optimum time of such therapy but discuss methods of restoring optimum normal endocrine function.

It should be noted that the longer a cycle lasts past the eight-week mark, the harder testosterone recovery becomes. The best way of gauging ones hormonal milieu and planning compensatory measures is to have blood tests done prior to and following cessation of AAS therapy. For the purpose of this Consensus Statement and the awareness of a lack of testing athletes, the following universally accepted post cycle hormone status is assumed:

a) Luteinizing Hormone (LH): low to none, Luteinizing Hormone Releasing Hormone (LHRH): low to none
b) Testosterone (T): low
c) Estrogen (E): high in relation to T
d) Cortisol (C): high
e) Red Blood Cell (RBC) count: falling


While all of these hormone measurements are assumed on the low end of the scale, biochemical individuality will ultimately determine where a person’s levels fall. So assumption of low to substandard levels will not always be true in everyone.


1. What are the goals of testosterone recovery?

The return of hormonal balance is but one goal of this program. To create a transitional period of minimized muscle loss and sustained and/or increased motivation is another.


2. Detailed Recommendations

If the athlete is ready to come off and is still taking long acting esters he shall switch to short acting drugs in order to have complete control of exogenous hormone levels. A “waiting period” for esters to clear is unacceptable and provides for a slow slide into the post cycle catabolic state. This period of short acting supplements shall last for a minimum of 2 weeks.

a) Luteinizing Hormone and shrunken testicles

H C G
If the testis have atrophied, the introduction of H C G at 1000iu x 14 days is necessary. To prevent this atrophy from happening, the use of H C G at 1000iu x 7 days every fourth week of the AAS cycle is recommended. This will provide exogenous LH and must only be used to restore/keep proper testicle size.
Week 1-2: H C G, 1000iu ed
C l o m i d
The practice of using Clomid at 50mg throughout the AAS cycle or 100mg a day for 3-5 days every 4th week has been used successfully to maintain proper testicle size

b) Low testosterone and lack of motivation

The introduction of exogenous hormones to compensate for the low endogenous testosterone levels may help to keep loss of drive, strength and muscle at bay but may also slow the recovery process. The below drug and application was chosen for its limited impact on the HPTA

D i a n a b o l
Studies and empirical evidence have shown Dianabol to be beneficial to keep Cortisol in check and provide some intermediate relief from the symptoms of low testosterone via an increase of dopamine, IGF-1, and Central Nervous System stimulation. The heightened dopamine will combat Prolactin and help raise the levels of endogenous Human Growth Hormone. Other studies point to a lack of LH suppression when taken first thing in the morning. It shall be noted that only a low dose upon rising is recommended in order to avoid further disruption of the HPTA
Week 1-6: 10mg dbol am, ed

c) High Estrogen and suppressed Hypothalamus- Pituitary- Testicular- Axis (HPTA)

Estrogen acts as the primary messenger of testosterone production. Testosterone is aromatized into estrogen, which signals the Hypothalamus to stop producing the proper testosterone release hormones. Estrogen must be kept low.

A r i m i d e x
A powerful aromatize inhibitor shall be part of every cycle. For testosterone recovery it is used to keep the testosterone/ estrogen balance in favor of testosterone. It is also of help to keep any additionally occurring estrogen from dbol and Androgel low to none. Studies have shown a 54% increase of testosterone in eugonadal patients
Week 1-10: ½-1mg ed
C l o m i d
Universally accepted as THE testosterone recovery tool. It blocks estrogen from the HPTA and stimulates the production of LHRH. LHRH then initiates the production of LH, which in turn signals the testis (if not atrophied) to produce testosterone.
Week 3-5: 100mg ed
Week 6-8: 50mg ed

d) High Cortisol, suppressed HPTA and catabolism

Cortisol is catabolic. It is the enemy of all anabolism and must be kept in check. While it is blocked when under the influence of AAS, it is free to attach to the Anabolic Receptors (AR) once the steroids leave. Due to this blockage Cortisol tends to accumulate and increase when on. A low level is desirable however since it is important for other vital functions such as control of inflammation. Balance is the key.

V i t a m i n C
At 3-5g before heavy workouts, it keeps the exercise induced rise of Cortisol in check
Always: 3-5g before workouts
D H E A
A useless pro-hormone as far as anabolism is concerned, this substance is great to keep Cortisol within normal levels. There is a correlation between high Cortisol and low DHEA levels.
Week 1-6: 150mg am and pm
H u m a l o g
It is well known that insulin possesses powerful anti Cortisol/anabolic properties, specially when used at times when Cortisol is high, such as early morning and post workout.
It is of utmost importance to be educated about insulin and its proper use. However, this CS defers to other available research material for more detailed recommendations and cautionary measures.
A minimum of 10g of dextrose/Maltodextrin per iu with a high carb/mixed glycemic index meal 45 min after insulin injection is suggested as a rough guide line for Humalog use only.
Perfect with dextrose/malto and Creatine.
Week 1-5: 10iu am and 10iu post workout
Caution: DO NOT EXCEED THESE RECOMMENDATIONS
D e x t r o s e a n d M a l to d e x t r i n
It is neither a supplement nor a drug, but these carbohydrates have a very high glycemic index and keep Cortisol levels low by increasing endogenous insulin or keep blood sugar normal when used with exogenous insulin. They also provide excellent energy for heavy workouts. In order to not gain unwanted fat, dextrose and/or maltodextrin shall be ingested during your workout and with your post workout shake only.
Always: 100g with workout water and 100g with post workout shake

e) Red Blood Cell Count and Stamina

E P O
Causes the bone marrow to increase red blood cell production and may have anabolic, fat burning and rejuvenating benefits.
It is of utmost importance to be educate about EPO and its proper use. However, this CS defers to other available research material for more detailed recommendations and cautionary measures.